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dc.creatorNahmod, Karen Amelia-
dc.creatorGeffner, Jorge Raúl-
dc.creatorWalther, Thomas-
dc.date2019-01-08T19:05:14Z-
dc.date2019-01-08T19:05:14Z-
dc.date2010-07-
dc.date2019-01-07T13:32:19Z-
dc.date.accessioned2019-04-29T15:50:09Z-
dc.date.available2019-04-29T15:50:09Z-
dc.date.issued2010-07-
dc.identifierNahmod, Karen Amelia; Geffner, Jorge Raúl; Walther, Thomas; Angiotensin II type 1a-deficient bone marrow-derived dendritic cells produce higher levels of monocyte chemoattractant protein 1; Lippincott Williams; Hypertension; 56; 7-2010; 6-7-
dc.identifier0194-911X-
dc.identifierhttp://hdl.handle.net/11336/67671-
dc.identifierCONICET Digital-
dc.identifierCONICET-
dc.identifier.urihttp://rodna.bn.gov.ar:8080/jspui/handle/bnmm/303302-
dc.descriptionWe read with interest the article from Crowley et al,1 who studied the role of angiotensin II type 1 (AT1) receptors (AT1aR) on immune cells in the pathogenesis of angiotensin II–induced hypertension by generating bone marrow chimeras with wild-type (WT) donors or donors lacking AT1aR. Interestingly, they found that the group of donors lacking AT1aR had more albuminuria and higher expression of a number of inflammatory mediators, including monocyte chemoattractant protein 1 (MCP-1), with persistent infiltration of macrophages in the kidney, concluding that AT1aR on bone marrow–derived cells had protective actions.-
dc.descriptionFil: Nahmod, Karen Amelia. Academia Nacional de Medicina de Buenos Aires. Instituto de Investigaciones Hematológicas "Mariano R. Castex"; Argentina. Consejo Nacional de Investigaciones Científicas y Técnicas; Argentina-
dc.descriptionFil: Geffner, Jorge Raúl. Academia Nacional de Medicina de Buenos Aires. Instituto de Investigaciones Hematológicas "Mariano R. Castex"; Argentina. Consejo Nacional de Investigaciones Científicas y Técnicas; Argentina-
dc.descriptionFil: Walther, Thomas. University of Hull; Reino Unido-
dc.formatapplication/pdf-
dc.formatapplication/pdf-
dc.languageeng-
dc.publisherLippincott Williams-
dc.relationinfo:eu-repo/semantics/altIdentifier/doi/http://dx.doi.org/10.1161/HYPERTENSIONAHA.110.153205-
dc.relationinfo:eu-repo/semantics/altIdentifier/url/https://www.ahajournals.org/doi/10.1161/HYPERTENSIONAHA.110.153205-
dc.rightsinfo:eu-repo/semantics/restrictedAccess-
dc.rightshttps://creativecommons.org/licenses/by-nc-sa/2.5/ar/-
dc.sourcereponame:CONICET Digital (CONICET)-
dc.sourceinstname:Consejo Nacional de Investigaciones Científicas y Técnicas-
dc.sourceinstacron:CONICET-
dc.subjectAT1 RECEPTOR-
dc.subjectANGIOTENSIN-
dc.subjectMCP-1-
dc.subjectDENDRITIC CELLS-
dc.subjectOtras Ciencias de la Salud-
dc.subjectCiencias de la Salud-
dc.subjectCIENCIAS MÉDICAS Y DE LA SALUD-
dc.titleAngiotensin II type 1a-deficient bone marrow-derived dendritic cells produce higher levels of monocyte chemoattractant protein 1-
dc.typeinfo:eu-repo/semantics/article-
dc.typeinfo:eu-repo/semantics/publishedVersion-
dc.typeinfo:ar-repo/semantics/articulo-
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